Summary of Study ST000614

This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR000449. The data can be accessed directly via it's Project DOI: 10.21228/M8161N This work is supported by NIH grant, U2C- DK119886.

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Study IDST000614
Study TitleTobacco-specific carcinogens in Bladder Cancer
Study SummarySmoking induced methylation plays a critical role in the accumulation of methylated metabolites, DNA adducts damage and altered metabolism in BCa. These deregulated metabolites can be detected non-invasively and can be used as causal biomarkers that can predict the risk of developing BCa in smokers
Institute
Baylor College of Medicine
Last NamePutluri
First NameNagireddy
AddressOne Baylor Plaza
Emailputluri@bcm.edu
Phone7137983144
Submit Date2017-05-23
Analysis Type DetailLC-MS
Release Date2018-12-11
Release Version1
Nagireddy Putluri Nagireddy Putluri
https://dx.doi.org/10.21228/M8161N
ftp://www.metabolomicsworkbench.org/Studies/ application/zip

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Project:

Project ID:PR000449
Project DOI:doi: 10.21228/M8161N
Project Title:Tobacco-specific carcinogens induce hypermethylation, DNA adducts and DNA damage in Bladder Cancer
Project Summary:Smoking is a major risk factor for the development of Bladder cancer (BCa); however, the metabolic alterations and functional consequences associated with cigarette smoke carcinogens in BCa remain poorly defined. We assessed the metabolic profile in BCa and identified the key alterations in metabolism associated with smoking.
Institute:Baylor College of Medicine
Last Name:Putluri
First Name:Nagireddy
Address:One Baylor Plaza, Houston, TX, 77030, USA
Email:putluri@bcm.edu
Phone:713-798-3139
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