Summary of project PR000969

This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench,, where it has been assigned Project ID PR000969. The data can be accessed directly via it's Project DOI: 10.21228/M8FD5Z This work is supported by NIH grant, U2C- DK119886.


Project ID: PR000969
Project DOI:doi: 10.21228/M8FD5Z
Project Title:Metabolic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice
Project Type:MS Analysis
Project Summary:Micronutrients are key regulators of prenatal one-carbon (C1) metabolism. In this study, we show here that prenatal excessive methionine (MET), a principal micronutrient and methyl-donor, produces in early life stages significant changes in the components of brain C1 pathways and other metabolic pathways including glutamate transmission, mitochondrial function, and lipid metabolism.
Institute:University of California, Irvine
Department:Department of Pharmaceutical Sciences, School of Medicine; Department of Computer Science, School of Information and Computer Sciences
Laboratory:Amal Alachkar Group; Pierre Baldi Group
Last Name:Chen
First Name:Siwei
Address:66304 Verano Place Rd. South

Summary of all studies in project PR000969

Study IDStudy TitleSpeciesInstituteAnalysis
(* : Contains Untargted data)
(* : Contains raw data)
ST001413 Metabolomic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice Mus musculus University of California, Irvine MS 2020-07-24 1 28 Uploaded data (2.1G)*