Compare metabolites in 2 of these studies:
Study A:   Study B:  

List of Studies ( Metabolite:LPC P-17:0)

Study_idAnalysis_idStudy_titleSourceSpeciesDiseaseInstituteAnalysis Type
ST003988 AN006569 Lipid and cell cycling perturbations driven by the HDAC inhibitor romidepsin render liver cancer vulnerable to RTK targeting and immunologically active Cultured cells Human Cancer CNRS LC-MS
ST003931 AN006455 Acylated putrescine therapeutic discovery for Inflammatory Bowel Diseases: HILIC-neg, C8-pos and C18-neg profiling of mouse fecal samples Feces Mouse Inflammatory bowel disease Broad Institute of MIT and Harvard LC-MS
ST003705 AN006078 Lipid Metabolite Profiling of nontuberculous mycobacterial pulmonary disease Blood Human Lung disease Seoul National University College of Medicine and Hospital LC-MS
ST003472 AN005707 An Optimized Plasmalogen Dietary Supplement Remodels the Cardiac Lipidome and Proteome, Providing Greater Protection in a Male Mouse Model of Dilated Cardiomyopathy Over Females Blood Mouse Heart disease Baker Heart and Diabetes Institute LC-MS
ST003472 AN005707 An Optimized Plasmalogen Dietary Supplement Remodels the Cardiac Lipidome and Proteome, Providing Greater Protection in a Male Mouse Model of Dilated Cardiomyopathy Over Females Heart Mouse Heart disease Baker Heart and Diabetes Institute LC-MS
ST003108 AN005089 Complete absence of GLUT1 does not impair human terminal erythroid differentiation Cultured cells Human GLUT1 Deficiency Syndrome University of Colorado LC-MS
ST003057 AN005012 Spatiotemporal mapping of lipid disturbance in heart injury - Part 1 Heart Mouse Heart injury National University of Singapore LC-MS
ST002939 AN004824 Role of PI3K in Atrial Myopathy: Insights from Transgenic Mouse Models and Identification of a Dysregulated PI3K Lipid Profile in Individuals with Atrial Fibrillation - Part 2 of 2, Mus musculus Atria Mouse Atrial fibrillation Baker Heart and Diabetes Institute LC-MS
ST002939 AN004824 Role of PI3K in Atrial Myopathy: Insights from Transgenic Mouse Models and Identification of a Dysregulated PI3K Lipid Profile in Individuals with Atrial Fibrillation - Part 2 of 2, Mus musculus Ventricles Mouse Atrial fibrillation Baker Heart and Diabetes Institute LC-MS
ST002938 AN004823 Role of PI3K in Atrial Myopathy: Insights from Transgenic Mouse Models and Identification of a Dysregulated PI3K Lipid Profile in Individuals with Atrial Fibrillation - part 1 of 2, human plasma Blood Human Atrial fibrillation Baker Heart and Diabetes Institute LC-MS
ST002403 AN003917 Deep multi-omic profiling reveals extensive mitochondrial remodeling driven by glycemia in early diabetic kidney disease (Mitochondria) Mitochondria Rat Kidney disease Baker Heart and Diabetes Institute LC-MS
ST002229 AN003638 Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice (Part 1) Blood Mouse Obesity Baker Heart and Diabetes Institute LC-MS
ST002229 AN003638 Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice (Part 1) Epididymal fat Mouse Obesity Baker Heart and Diabetes Institute LC-MS
ST002229 AN003638 Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice (Part 1) Liver Mouse Obesity Baker Heart and Diabetes Institute LC-MS
ST002229 AN003638 Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice (Part 1) Muscle Mouse Obesity Baker Heart and Diabetes Institute LC-MS
ST002229 AN003638 Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice (Part 1) Ventricles Mouse Obesity Baker Heart and Diabetes Institute LC-MS
ST001907 AN003104 Training-induced bioenergetic improvement in human skeletal muscle is associated with non-stoichiometric changes in the mitochondrial proteome without reorganisation of respiratory chain content Muscle Human Baker Heart and Diabetes Institute LC-MS
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