Compare metabolites in 2 of these studies:
Study A:   Study B:  

List of Studies ( Metabolite:Prosopinine)

Study_idAnalysis_idStudy_titleSourceSpeciesDiseaseInstituteUnits(range)
ST001795 AN002915 Changes in mesenteric lymph lipid profile of mice upon high-fat diet with and without Celecoxib (part I) Mesenteric lymph Mouse Obesity Monash Institute of Pharmaceutical Sciences height
ST001788 AN002900 β-Adrenergic regulation of metabolism in macrophages (part-IV) Macrophages Human Monash University Intensity
ST000148 AN000235 A549 13C-labeled Cell Study Epithelial cells Human Cancer University of Kentucky normalized corrected peak area
ST000142 AN000225 H1299 13C-labeled Cell Study Lung Human Cancer University of Kentucky Peak area
ST000612 AN000937 Sphingolipid Analysis of Human Aqueous Humor in Glaucomatous and Control eyes Eye tissue Human Eye disease University of Miami Peak area
ST000613 AN000938 Human TM Sphingolipid Analysis (part II) Eye tissue Human Eye disease University of Miami Peak area
ST000620 AN000952 Sphingolipid Analysis of hyper and normotensive DBA2J mice aqueous humor and trabecular meshwork Mouse Eye disease University of Miami Peak area
ST002792 AN004542 Chemoproteomics validates selective targeting of Plasmodium M1 alanyl aminopeptidase as a cross-species strategy to treat malaria Blood Plasmodium falciparum Malaria Monash University peak height
ST000549 AN000837 Investigating large scale metabolomics in mice serum lacking insulin receptors and IGF-1 receptors Blood Mouse Diabetes Mayo Clinic Peak intensity
ST002094 AN003420 Commensal intestinal microbiota regulates host luminal proteolytic activity and intestinal barrier integrity through β-glucuronidase activity (Part 1) Feces Human Irritable bowel syndrome Mayo Clinic raw intensity
ST002094 AN003421 Commensal intestinal microbiota regulates host luminal proteolytic activity and intestinal barrier integrity through β-glucuronidase activity (Part 1) Feces Human Irritable bowel syndrome Mayo Clinic raw intensity
ST001175 AN001950 Multi-omics analysis demonstrates unique mode of action of a potent new antimalarial compound, JPC-3210, against Plasmodium falciparum Plasmodium cells Plasmodium falciparum Malaria Monash University Signal Intensity
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