Summary of Study ST000614
This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR000449. The data can be accessed directly via it's Project DOI: 10.21228/M8161N This work is supported by NIH grant, U2C- DK119886.
See: https://www.metabolomicsworkbench.org/about/howtocite.php
Study ID | ST000614 |
Study Title | Tobacco-specific carcinogens in Bladder Cancer |
Study Summary | Smoking induced methylation plays a critical role in the accumulation of methylated metabolites, DNA adducts damage and altered metabolism in BCa. These deregulated metabolites can be detected non-invasively and can be used as causal biomarkers that can predict the risk of developing BCa in smokers |
Institute | Baylor College of Medicine |
Last Name | Putluri |
First Name | Nagireddy |
Address | One Baylor Plaza |
putluri@bcm.edu | |
Phone | 7137983144 |
Submit Date | 2017-05-23 |
Analysis Type Detail | LC-MS |
Release Date | 2018-12-11 |
Release Version | 1 |
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Project:
Project ID: | PR000449 |
Project DOI: | doi: 10.21228/M8161N |
Project Title: | Tobacco-specific carcinogens induce hypermethylation, DNA adducts and DNA damage in Bladder Cancer |
Project Summary: | Smoking is a major risk factor for the development of Bladder cancer (BCa); however, the metabolic alterations and functional consequences associated with cigarette smoke carcinogens in BCa remain poorly defined. We assessed the metabolic profile in BCa and identified the key alterations in metabolism associated with smoking. |
Institute: | Baylor College of Medicine |
Last Name: | Putluri |
First Name: | Nagireddy |
Address: | One Baylor Plaza, Houston, TX, 77030, USA |
Email: | putluri@bcm.edu |
Phone: | 713-798-3139 |